Nursing School
Herpes Zoster (Shingles) Nursing Care Plan
Shingles is reactivated varicella zoster virus (VZV), the same virus behind chickenpox. After chickenpox resolves, VZV stays dormant in the spinal nerve gangl…
Medically reviewed by Jonathan Kim, DO
Last reviewed Jun 11, 2026·Next review Jun 11, 2027
care-plan
Shingles is reactivated varicella zoster virus (VZV), the same virus behind chickenpox. After chickenpox resolves, VZV stays dormant in the spinal nerve ganglia, then reactivates and travels down the peripheral nerves to the skin, where it erupts in painful vesicles. It hits older adults and immunocompromised patients hardest. On the floor your focus is pain control, infection prevention, contact isolation, and teaching, because the pain can outlast the rash as postherpetic neuralgia.
VZV usually tracks the trunk but can appear on the buttocks or face. If it involves the ophthalmic nerve, the patient risks keratitis, ulceration, and even blindness. Secondary infection from scratching is common.
A patient is infectious for the first 2 to 3 days after eruption. Incubation runs 7 to 21 days. The full course is 10 days to 5 weeks from onset to recovery. Some patients develop postherpetic neuralgia that lingers well after the lesions heal.
Shingles starts with burning, tingling, numbness, or itch in the affected area. VZV can involve the central nervous system (CNS), and pneumonia develops in about 15% of cases. Roughly 20% of people who have had chickenpox go on to develop shingles.
Nursing Care Plans and Management
Main goals: the patient understands the disease and treatment, gets relief from the lesions, stays in strict contact isolation, develops self-acceptance, and avoids complications.
Nursing Problem Priorities
- Manage acute pain and discomfort.
- Minimize complications and infection.
- Promote healing and prevent scarring.
- Teach self-care.
- Support emotional wellbeing.
- Prevent transmission.
- Provide followup and monitoring.
Nursing Assessment
Assess for these subjective and objective findings:
- Altered muscle tone
- Facial mask of pain
- Burning, dull, or sharp pain
- Pain localized to the affected nerve
- Poor followup of instructions
- Repeated questioning of the care team
- Verbalizing inaccurate information
Assess for related factors:
- Nerve pain (commonly cervical, lumbar, sacral, thoracic, or the ophthalmic division of the trigeminal nerve)
- Complex treatment regimen
- Emotional state affecting learning
- Herpes zoster outbreak
- New condition and procedures
- Crusted-over lesions
- Itching and scratching
- Skin lesions (papules, vesicles, pustules)
- Preoccupation with the changed body part
- Visible skin lesions
Nursing Diagnosis
Form the diagnosis from your assessment and clinical judgment, matching the plan to this patient's pain, skin, and risk of transmission rather than to a label.
Nursing Goals
- The patient rests comfortably.
- The patient reports pain controlled to less than 3 to 4 on a 0 to 10 scale.
- The patient or caregiver states the key information about the disease, signs and symptoms, treatment, and complications.
- The patient stays free of secondary infection, shown by intact skin without redness or lesions.
- The patient has minimal transmission risk through universal precautions.
- The patient voices feelings about the lesions and keeps up daily activities.
- The patient shows positive body image, able to look at, talk about, and care for the lesions.
Nursing Interventions and Actions
1. Managing Acute Pain
Shingles pain can be excruciating and long-lasting. Controlling it matters for comfort and for heading off chronic pain such as postherpetic neuralgia (PHN).
Assess the pain: severity, location, quality, duration, and what triggers or relieves it. Patients describe tingling, burning, or extreme hyperesthesia in one skin area, often days before lesions appear. PHN is chronic pain that can persist after lesions heal, either constant or intermittent.
Watch for nonverbal signs of pain. Pain thresholds and expression vary, and some patients deny pain that is clearly there. Associated signs help you read it.
Teach these comfort measures:
- Wear loose cotton clothing. Tight, nonbreathing fabric rubs lesions; cotton lets moisture evaporate.
- Apply cool, moist dressings to itchy lesions, with or without Burrow's solution, several times a day. Stop once the lesions dry. This relieves itch and lowers secondary infection risk.
- Avoid temperature extremes in air and bathwater. Tepid water itches and burns the least.
- Don't rub or scratch the skin or lesions. Scratching increases itch and the chance of secondary infection.
- Use topical steroids (anti-inflammatory), antihistamines (anti-itch, useful at bedtime), and analgesics. Relief often takes a combination.
Give medications as ordered. Oral opioids (codeine, hydrocodone) are typical in the acute phase. Analgesics, antidepressants, and antiepileptics manage PHN. Topical options for PHN include capsaicin cream (Zostrix) and lidocaine-prilocaine cream (EMLA).
2. Infection Control and Prevention
Assess for skin lesions and their location. Lesions are fluid-filled, turn yellow, then crust over, on one side of the trunk or buttock. They follow dermatomes in band-like strips and can appear on the face, arms, and legs when those nerves are involved. As they rupture and crust, they look like chickenpox lesions.
Assess for pruritus, irritation, and how much the patient scratches. Look for localized infection: redness and drainage. Scratching opens pustules and introduces bacteria.
Check for lesions around the eye or ear. The virus can seriously damage the eyes and ears, causing blindness or hearing loss. To find corneal lesions, the provider stains the cornea with fluorescein and views it under a Wood's lamp.
Assess the patient's and family's immunization status and chickenpox history. Patients with shingles are contagious to anyone who has not had chickenpox. Those who have had varicella vaccine are considered immune but should have varicella titers to confirm.
Get culture and sensitivity of suspected infected lesions as ordered. This guides antibiotic choice.
Get additional cultures and bloodwork as indicated. Viral culture, Tzanck smear, or viral smear may be needed for diagnosis, along with serology.
Teach contact isolation. VZV spreads through contact with fluid from the lesions.
Tell the patient to stay away from pregnant women and immunocompromised people. Active lesions are infectious, and those groups are more vulnerable.
Use universal precautions. VZV can spread and cause chickenpox in anyone who has not had it.
Use gauze to separate lesions in skin folds. This cuts irritation, itching, and cross-contamination.
Discourage scratching and have the patient trim fingernails. These measures prevent the inadvertent opening of lesions, cross-contamination, and bacterial infection.
Teach antiviral use as prescribed. Antivirals work best in the first 72 hours of an outbreak while the virus is replicating. Drugs of choice are acyclovir, famciclovir, or valacyclovir.
Teach systemic steroid use if ordered for anti-inflammatory effect. Their use is controversial and mostly reserved for severe cases.
3. Improving Body Image
Painful, visible lesions can hit body image hard, especially for patients who already carry appearance concerns.
Assess how the patient sees the change in appearance. An outbreak can last weeks while the patient keeps working and living normally, so they may need help coping with the change.
Note comments about the lesions. Repeated outbreaks or infected lesions can scar, which feeds preoccupation with appearance.
Explain the reasons for infectious isolation. Sitting down to talk and listen in the room eases the isolation and loneliness.
Help the patient prepare answers about the lesions and infection risk. Rehearsing responses to expected questions can reassure them.
Suggest concealing clothing when lesions are easy to cover. This helps patients struggling with body image changes.
4. Patient Education and Health Teaching
Check what the patient and caregiver understand about the disease, treatment, and complications. An underlying illness may have weakened the patient and let the zoster surface, and they should understand that.
Find out whether the caregiver or family has had chickenpox or varicella vaccine, or is immunocompromised. Even though varicella vaccine does not confer immunity to shingles, shingles is less common in vaccinated adults than in those who had chickenpox.
Give the patient and caregiver written information. Patients often confuse herpes zoster with genital herpes. Clarify it, since they may be reluctant to ask. They need a full understanding to take part in their care.
- Why isolation matters. Isolate clothing and linen, including towels.
- What shingles is and how it spreads. Lesion fluid carries the virus and spreads by direct contact.
- When to call the care team about CNS inflammation (changes in level of consciousness). Early assessment means prompt treatment of complications.
Encourage shingles vaccination. Shingrix (recombinant zoster vaccine) is the recommended vaccine for adults 50 years and older, given as 2 doses 2 to 6 months apart. It is preferred over the older Zostavax, which was discontinued in the United States in 2020. Shingrix is about 97% effective at preventing shingles in adults 50 to 69 years old and about 91% effective in adults 70 and older. It is not given to anyone with a known allergy to its components.
5. Pharmacologic Support
Antivirals, analgesics, and adjuncts manage pain, slow viral replication, and lower PHN risk.
Antivirals
- Acyclovir. Inhibits viral replication, shortens and softens the rash, and helps prevent or ease PHN.
- Valacyclovir. Converts to acyclovir in the body, with similar antiviral effect.
- Famciclovir. Converts to penciclovir, which inhibits viral replication.
Analgesics
- NSAIDs. Ibuprofen and naproxen for mild to moderate pain.
- Opioids. Oxycodone or tramadol, cautiously and short-term, for severe pain.
Tricyclic antidepressants
- Amitriptyline. For neuropathic pain, especially PHN.
- Nortriptyline. Similar to amitriptyline for nerve pain.
Anticonvulsants
- Gabapentin. Eases nerve pain and lowers PHN risk.
- Pregabalin. For neuropathic pain associated with shingles.
Antibiotics
- For bacterial superinfection from scratching or broken skin.
6. Monitoring Diagnostics and Labs
Viral culture. A sample from the lesions or vesicle fluid goes to the lab to isolate and identify VZV.
Polymerase chain reaction (PCR). Highly sensitive and specific for VZV genetic material. Run on lesion samples, cerebrospinal fluid (CSF), or blood to confirm the virus and tell it apart from look-alike infections.
Tzanck smear. A sample from the base of a vesicle, stained to show multinucleated giant cells. It supports the diagnosis but does not separate varicella from zoster.
Blood tests. Check immune status, inflammation, and rule out other causes:
- Complete blood count (CBC). The white blood cell count points to infection or inflammation.
- C-reactive protein (CRP). Elevated levels suggest inflammation.
- VZV antibody testing. Detects IgG and IgM against VZV to gauge immune status and tell recent infection from past exposure.
Lumbar puncture (spinal tap). Done when CNS involvement is a concern. CSF is checked for VZV DNA, abnormal cell counts, and protein.
7. Monitoring for Complications
Most shingles resolves cleanly, but immunocompromised and older patients are at higher risk for complications. Catch them early.
Assess pain regularly with a validated scale (e.g., numerical rating scale) to track PHN. Pain is a common complication, and tracking it shows whether your interventions work.
Teach the signs of ocular complications (if the ophthalmic division is involved) and to seek immediate ophthalmology evaluation for any abnormality. Eye involvement can cause serious damage, and early treatment prevents vision loss.
Monitor and document changes in visual acuity, eye redness, swelling, or eye pain. These may signal ophthalmic complications that need prompt ophthalmology care.
Do regular neuro assessments for CNS involvement: headache, altered mental status, focal deficits. Neurologic complications are rare but need early recognition to prevent further damage.
Watch the rash for bacterial infection: increased redness, warmth, swelling, or purulent discharge. Superimposed infection needs prompt antibiotics.
Assess for disseminated zoster: new lesions in other areas or systemic symptoms like fever and malaise. This is more likely in immunocompromised patients, and early recognition lets you start antiviral therapy before severe illness.
Teach hygiene: keep the area clean and dry, avoid scratching, use recommended topicals. This prevents secondary infection and supports healing.
Offer emotional support and education about the complications, their likely duration, and managing discomfort. Shingles is distressing, and support improves understanding and adherence.